论文标题
相对论辐射介导的冲击中的血浆动力学作用
Plasma kinetic effects in relativistic radiation mediated shocks
论文作者
论文摘要
在光学较厚的介质中形成的快速冲击是由康普顿散射介导的,如果相对论是成对的。由于辐射力主要作用于电子和正电子,因此提出的问题是如何将力介导的离子是电击能的主要载体。人们普遍认为,辐射力诱导的小电荷分离会在减速离子的冲击中产生电场。在本文中,我们认为,虽然在没有正电子的亚相关性冲击中是正确的,但相对论辐射介导的冲击(RRMS)以新创建的$ E^+E^ - $对主导,但由于对电子和poSitrons的相反电力而需要其他耦合,但需要附加耦合。具体而言,我们表明,离子能量的耗散必须涉及集体等离子体相互作用。通过构建结合摩擦力的RRM的多流体模型,我们估计电子和正电子(和/或离子)之间的动量转移是通过在数十个比例到数千个质子皮肤深度的尺度上的集体相互作用(和/或离子)在$ e^+e^+e^ - $ pairs和离子之间的耗时和既适合于均等的。这留下了一个问题,问题是否可以通过散射血浆湍流来有效地加速到高能。这种加速可能对相对论的冲击突破信号产生重要影响。
Fast shocks that form in optically thick media are mediated by Compton scattering and, if relativistic, pair creation. Since the radiation force acts primarily on electrons and positrons, the question arises of how the force is mediated to the ions which are the dominant carriers of the shock energy. It has been widely thought that a small charge separation induced by the radiation force generates electric field inside the shock that decelerates the ions. In this paper we argue that, while this is true in sub-relativistic shocks which are devoid of positrons, in relativistic radiation mediated shocks (RRMS), which are dominated by newly created $e^+e^-$ pairs, additional coupling is needed, owing to the opposite electric force acting on electrons and positrons. Specifically, we show that dissipation of the ions energy must involve collective plasma interactions. By constructing a multi-fluid model for RRMS that incorporates friction forces, we estimate that momentum transfer between electrons and positrons (and/or ions) via collective interactions on scales of tens to thousands proton skin depths, depending on whether friction is effective only between $e^+e^-$ pairs or also between pairs and ions, is sufficient to coupe all particles and radiation inside the shock into a single fluid. This leaves open the question whether in relativistic RMS particles can effectively accelerate to high energies by scattering off plasma turbulence. Such acceleration might have important consequences for relativistic shock breakout signals.