论文标题

经济与疾病的传播:共同开放机制19

Economy Versus Disease Spread: Reopening Mechanisms for COVID 19

论文作者

Augustine, John, Hourani, Khalid, Molla, Anisur Rahaman, Pandurangan, Gopal, Pasic, Adi

论文摘要

我们研究了重新开放经济活动的机制,这些机制探讨了包含Covid-19的传播与最大化经济影响之间的权衡。由于许多组织,城市和州正在制定重新开放策略,这是当前重要的。我们的机制(称为小组调度)是基于将人口分为组,并在适当的日子安排每个小组的可能差距(当一切都被隔离时)。每个小组都没有与其他群体互动,重要的是,任何有症状的人都会被隔离。具体而言,我们的机制的特征是三个参数$(g,d,t)$,其中$ g $是组的数量,$ d $是连续安排组的天数,而$ t $是周期之间的差距。我们表明,我们的机制有效地将经济活动进行了更有效的控制,以更有效地控制Covid-19病毒。特别是,我们表明$(2,5,0)$机制将人口分为两组,分别有效五天,固定了相当有效的19例案件的数量,同时仍将经济活动保持在前期前19级的70%。我们还研究诸如$(2,3,2)$和$(3,3,0)$之类的机制,这些机制以较低的经济产出(约50%)为例,以对病毒的更具积极控制为代价;这些可能适用于疾病传播在人群中更加严重的情况下。我们通过理论分析和各种流行病学模型的广泛实验模拟来证明我们机制的功效。我们的机制仅通过调节人类的互动而证明是有益的。此外,我们的结果表明,如果通过遵循社会距离,戴口罩和其他公共卫生指南来使疾病传播(生殖)率降低,则它可以进一步提高我们机制的功效。

We study mechanisms for reopening economic activities that explore the trade off between containing the spread of COVID-19 and maximizing economic impact. This is of current importance as many organizations, cities, and states are formulating reopening strategies. Our mechanisms, referred to as group scheduling, are based on partitioning the population into groups and scheduling each group on appropriate days with possible gaps (when all are quarantined). Each group interacts with no other group and, importantly, any person who is symptomatic in a group is quarantined. Specifically, our mechanisms are characterized by three parameters $(g,d,t)$, where $g$ is the number of groups, $d$ is the number of days a group is continuously scheduled, and $t$ is the gap between cycles. We show that our mechanisms effectively trade off economic activity for more effective control of the COVID-19 virus. In particular, we show that the $(2,5,0)$ mechanism, which partitions the population into two groups that alternatively work for five days each, flat lines the number of COVID-19 cases quite effectively, while still maintaining economic activity at 70% of pre-COVID-19 level. We also study mechanisms such as $(2,3,2)$ and $(3,3,0)$ that achieve a somewhat lower economic output (about 50%) at the cost of more aggressive control of the virus; these could be applicable in situations when the disease spread is more rampant in the population. We demonstrate the efficacy of our mechanisms by theoretical analysis and extensive experimental simulations on various epidemiological models. Our mechanisms prove beneficial just by regulating human interactions. Moreover, our results show that if the disease transmission (reproductive) rate is made lower by following social distancing, mask wearing, and other public health guidelines, it can further increase the efficacy of our mechanisms.

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